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Heart Failure


Patients who receive target or near-target doses of evidence-based medicine (EBM) are more likely to continue working after a heart failure hospitalization, according to a study of Danish individuals published Dec. 6 in JACC: Heart Failure.

Damage inflicted during non-cardiac surgery can reach heart cells and significantly raise a patient’s risk of mortality for up to one year after the procedure, according to research published this week in the American Heart Association journal Circulation.

Biomedical engineers at Duke University have created a fully functioning heart muscle from pluripotent stem cells that is large enough to patch over damaged areas in human heart attack patients.

Heart failure-related hospitalizations and in-hospital mortalities have decreased significantly since the turn of the millennium, the American Heart Association has reported, despite a general increase in the burden of heart failure (HF) comorbidities.

The number of new people diagnosed with heart failure each year in the United Kingdom is now similar to that of the most four common types of cancer combined, according to the most comprehensive review of heart failure statistics in the U.K. to date, published online Nov. 21 in The Lancet.


Recent Headlines

Cooling cardiac arrest patients for 48 hours could be beneficial

Physicians have long used cooling methods to help patients wake up after suffering a cardiac arrest, and researchers from Aarhus University in Denmark are exploring how to make the approach even more effective.

Big promises? Questionable stem cell therapies marketed to heart failure patients

In a research letter published online July 24 in JAMA Internal Medicine, four physicians from the St. Louis University contacted 61 centers offering stem cell therapy, which is not approved by the FDA, to heart failure patients.

LSU research paves way for treatment to prevent brain damage after cardiac arrest

New research from Louisiana State University (LSU) in New Orleans could lead to a treatment that prevents long-term sensory problems that arise from brain damage that can occur in survivors of cardiac arrest.

3 U.S. airports now offer CPR training kiosks

Three airports around the U.S. have joined an American Heart Association initiative to provide hands-only CPR training kiosks for passengers waiting for flights.

Even minor weight gain could cause heart failure

Physicians have long warned patients against gaining significant weight, telling them that too much could cause cardiovascular disease. But new research shows that even a little weight, as little as 5 percent, could make a difference in one’s heart health.

Heart attack patients don’t adhere to smoking cessation prescriptions

In a new study, researchers at Duke University set out to explore how many patients who regularly smoke and are hospitalized for a heart attack receive cessation medications at discharge.

Complex med regimens lead to nonadherence in heart failure patients

A team from the University of Colorado (UC) in Boulder and Vanderbilt Heart and Vascular Institute in Tennessee published a study of the impact of polypharmacy in patients with heart failure in Clinical Interventions in Aging.

Research presented at APSC reveals early-onset heart failure in Asia

In a series of late-breaking trials being presented at the Asian Pacific Society of Cardiology’s (APSC) meeting in Singapore, research is surfacing about the realities of heart failure in Asian countries.

Study suggests first cardiac events more fatal for black patients

A new study conducted by researchers at Weill Cornell Medical College in New York shows that black patients are twice as likely as white counterparts to die from an initial heart event, suggesting heart attack prevention among blacks should be emphasized.

Researchers pinpoint why some don't respond to beta-blockers

New research may show why some heart failure patients don’t respond to beta-blocker drugs—with the cause lying in the dysfunction of beta-adrenergic receptor 3 (β3AR) and resulting decreases in cardioprotective phospholipid.